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With that in mind, it would make sense to use a painkiller that can do both of these. Aspirin and NSAIDS (non-steroidal anti-inflammatory drugs), such as ibuprofen and Motrin decrease prostaglandins. This can result in decreased pain and inflammation, especially if prostaglandins are main causes.
However, aspirin and NSAIDS do not directly affect other pain chemicals and do not affect pain nerve, C fiber.
Narcotics, such as Darvon or codeine, have no known effect on either "pain" bio-chemicals or pain nerves. Acetaminophen (Tylenol) also has no significant effect on these at usual doses. And in fact, way acetaminophen works is not actually known.
But we also know we can interrupt pain impulse away from injury site, at spinal cord. If our pain killer could also decrease release of (or deplete C fiber of) Substance P, pain impulse would be blocked at spinal cord level. Aspirin and NSAIDS have no known effect at this site. Narcotics and tricyclic antidepressants, such as amitriptylene or Elavil, on other hand, actually can block release of Substance P and stop transmission at spinal cord level, but once again have no value in decreasing levels of bio-chemicals of pain.
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Jaynne is an instructor at the local community college. She is also a former skipatroller and thus very interested in health issues. Read her latest article right here: